pediagenosis
Article Update
Loading...

Wednesday, June 16, 2021

Maxillary Artery Anatomy

Maxillary Artery Anatomy

Maxillary Artery Anatomy

Maxillary Artery Anatomy


1. Sphenopalatine artery

2. Posterior superior alveolar artery

3. Descending palatine artery in pterygopalatine fossa

4. Inferior alveolar artery

5. Middle meningeal artery

6. Deep temporal arteries and nerves

Carotid Arteries Anatomy

Carotid Arteries Anatomy

Carotid Arteries Anatomy

Carotid Arteries Anatomy


1. Superficial temporal artery

2. Occipital artery

3. Internal carotid artery

4. External carotid artery

5. Common carotid artery

6. Superior thyroid artery and Superior laryngeal branch

7. Ascending pharyngeal artery

8. Lingual artery

9. Facial artery

10. Posterior auricular artery

11. Maxillary artery

Subclavian Artery Anatomy

Subclavian Artery Anatomy

Subclavian Artery Anatomy

Subclavian Artery Anatomy


1. Vertebral artery

2. Costocervical trunk

3. Supreme intercostal artery

4. Internal thoracic artery

5. Suprascapular artery

6. Thyrocervical trunk

7. Common carotid artery

8. Transverse cervical artery

9. Inferior thyroid artery

Tuesday, June 15, 2021

General Anesthetics: Properties

General Anesthetics: Properties

General Anesthetics: Properties

General Anesthetics: Properties


General anesthetics (inhalational and intravenous agents) have a rapid, smooth onset of action and clinically desirable rapid reversal of effect. Concentrations of inhalational agents in the body and the pharmacokinetics depend on the drugs’ partial pressure in the lungs and solubility in blood and brain tissue. Induction of anesthesia is more rapid for drugs with high partial pressure in the lungs and high solubility in blood (eg, nitrous oxide, desflurane, sevoflurane). Onset of anesthesia is slowed when pulmonary blood flow is reduced. 

Local Anesthetics: Spinal Afferents and Local Anesthetic Mechanisms of Action

Local Anesthetics: Spinal Afferents and Local Anesthetic Mechanisms of Action

Local Anesthetics: Spinal Afferents and Local Anesthetic Mechanisms of Action

Local Anesthetics: Spinal Afferents and Local Anesthetic Mechanisms of Action


Local anesthetics cause temporary loss of pain sensation without loss of consciousness by blocking conduction along sensory nerve fibers. Some selectivity for pain afferents is achieved partly by using the agent close to target neurons. All currently used drugs block voltage­dependent Na+ channels in excitable cells, which decreases the likelihood of an action potential. The target site of the drugs is on the cytoplasmic side of the neuron membrane, so drug molecules must pass through the membrane.

Pain Pathways

Pain Pathways

Pain Pathways

Pain Pathways


Tissue injury can lead to cellular changes involving release of chemicals (eg, histamine) that start or quicken neuronal impulses that are interpreted as pain. Many neuronal pathways transmit pain sensation. For example, pain from peripheral injury reaches the CNS via primary afferent neurons, whose cell bodies form the DRG. 

Motor Neurons and Drugs

Motor Neurons and Drugs

Motor Neurons and Drugs

Motor Neurons and Drugs


Skeletal muscle spasticity often results from neuronal, not muscle, deficits. The reflex arc involved in coordinated skeletal muscle action involves several neurons, including interneurons, in the spinal cord. These spinal polysynaptic reflex arcs are depressed by a number of drugs, including barbiturates. However, nonspecific depression of synapses is not desirable because normal muscle function can be disrupted. 

Anatomy Physiology

[AnatomyPhysiology][recentbylabel2]

Featured

[Featured][recentbylabel2]
Notification
This is just an example, you can fill it later with your own note.
Done