pediagenosis
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Wednesday, September 16, 2026

Shoulder with Details of Glenohumeral Joint Anatomy

Shoulder with Details of Glenohumeral Joint Anatomy


Shoulder with Details of Glenohumeral Joint Anatomy


Shoulder with Details of Glenohumeral Joint Anatomy
Anterior view, Acromion, Coracoacromial ligament, Supraspinatus tendon (cut), Coracohumeral ligament, Greater tubercle, Transverse humeral ligament, Intertubercular tendon sheath (communicates with synovial cavity), Acromioclavicular joint capsule (incorporating acromioclavicular ligament), Subscapularis tendon (cut), Biceps brachii tendon (long head), Clavicle, Trapezoid ligament Coracoclavicular, Conoid ligament ligament, Superior transverse scapular ligament and superior scapular (suprascapular) notch, Coracoid process, Opening of subtendinous bursa of subscapularis muscle, 

Tuesday, September 15, 2026

Monday, September 14, 2026

Diagnosis of Death: Circulatory Death, Brainstem Death, and Physiology

Diagnosis of Death: Circulatory Death, Brainstem Death, and Physiology


Diagnosis of death showing brainstem death, circulatory death, Cushing reflex, catecholamine storm, and neuroendocrine changes



Diagnosis Of Death And Its Physiology
Diagnosing death
Circulatory death
Traditionally, death has been certified by the absence of a circulation, usually taken as the point at which the heart stops beating. In the UK, current guidance suggests that death may be confirmed after 5 minutes of observation following cessation of cardiac function (e.g. absence of heart sounds, absence of palpable central pulse or asystole on a continuous electrocardiogram). Organ donation after circulatory death (DCD) may occur following confirmation that death has occurred (also called non-heart-beating donation). There are two sorts of DCD donation, controlled and uncontrolled.

Thursday, September 10, 2026

Myxedema: Symptoms, Causes, Skin Findings, Diagnosis & Treatment

Myxedema: Symptoms, Causes, Skin Findings, Diagnosis & Treatment



Myxedema skin findings showing periorbital edema, dry skin, facial changes, and other clinical features of severe hypothyroidism





MYXEDEMA
Myxedema is seen in patients with untreated severe hypothyroidism. This condition results from a total lack of thyroid hormone secretion and resultant deposition of mucopolysaccharides into the skin and other organs. Many skin and systemic findings are present in severe hypothyroidism. This is a condition seen in adults. The infantile form, called cretinism, is still found in parts of the world that do not routinely test newborn infants. If it is left untreated, mental retardation and various neurological deficits can occur. Adult myxedema is an uncommon clinical disease.

Wednesday, September 9, 2026

Cross Section at L3–4 Anatomy

Cross Section at L3–4 Anatomy

Cross Section at L3–4 Anatomy




Cross Section at L3–4 Anatomy
Round ligament (ligamentum teres) of liver, Transverse colon, Branches of inferior epigastric vessels, Rectus sheath, Transversus abdominis aponeurosis, External oblique aponeurosis, Internal oblique aponeurosis, Mesentery of small intestine, Superior mesenteric vessels, Small intestine (ileum), Lymph node, Ascending colon, Right paracolic gutter, Tendon of origin of transversus abdominis muscle, Ilioinguinal nerve,

Tuesday, September 8, 2026

Dermatomyositis: Symptoms, Causes, Diagnosis, Histology & Treatment

Dermatomyositis: Symptoms, Causes, Diagnosis, Histology & Treatment

Dermatomyositis with characteristic skin rash and proximal muscle weakness



Dermatomyositis
Dermatomyositis is a chronic connective tissue disease that can be associated with an underlying internal malignancy. This connective tissue disease shares similarities with polymyositis, but the latter has no cutaneous findings. Up to one third of patients with dermatomyositis have an underlying malignancy. The myositis is often prominent and manifests as tenderness and weakness of the proximal muscle groups. The pelvic and shoulder girdle muscles are the ones most commonly affected. Dermatomyositis sine myositis is a well-recognized variant that has only the cutaneous findings; evidence of muscle involvement is absent.

Monday, September 7, 2026

Topography and Constrictions of Esophagus Anatomy

Topography and Constrictions of Esophagus Anatomy


Topography and Constrictions of Esophagus Anatomy


Topography and Constrictions of Esophagus Anatomy
Incisor tooth, Oropharynx, Epiglottis, Piriform recess, Thyroid cartilage, Cricoid cartilage, Thyroid cartilage, Cricoid cartilage, Cricopharyngeus (muscle) part of inferior pharyngeal constrictor muscle, Pharyngoesophageal constriction, Average length in centimeters, Thoracic (aortobronchial) constriction, Trachea, Arch of aorta, Left main bronchus,
Alport Syndrome and Thin Basement Membrane Nephropathy: Causes, Symptoms, Diagnosis & Treatment

Alport Syndrome and Thin Basement Membrane Nephropathy: Causes, Symptoms, Diagnosis & Treatment

Alport syndrome and thin basement membrane nephropathy showing type IV collagen defects in the glomerular basement membrane


HEREDITARY NEPHRITIS (ALPORT SYNDROME)/THIN BASEMENT MEMBRANE NEPHROPATHY
Hereditary nephritis (HN, also known as Alport syndrome) and thin basement membrane nephropathy (TBMN) are both inherited disorders that feature structural defects in type IV collagen, an integral component of the glomerular basement membrane. Both conditions present in childhood with persistent microscopic hematuria.
HN is rare, affecting 1 in 50,000 individuals, and often progresses to end stage renal disease (ESRD). TBMN, in contrast, affects 1 in 20 to 100 individuals and typically does not have a progressive course.

Sunday, September 6, 2026

Intracellular Accumulations: Causes, Types, Examples, and Clinical Significance

Intracellular Accumulations: Causes, Types, Examples, and Clinical Significance

Intracellular accumulations showing lipid, glycogen, lipofuscin, bilirubin, and exogenous pigment buildup in cells




Intracellular Accumulations.

Intracellular accumulations represent the buildup of substances that cells cannot immediately use or eliminate. The substances may accumulate in the cytoplasm (frequently in the lysosomes) or in the nucleus. In some cases the accumulation may be an abnormal substance that the cell has produced, and in other cases the cell may be storing exogenous materials or products of pathologic processes occurring elsewhere in the body. An example would be the accumulation of beta amyloid fragments, which progress to a skeletal muscle disorder called myositis. These substances may  accumulate  transiently  or  permanently, and they may be harmless or, in some cases, toxic.

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